[CitizensTruth] flu vaccine and nerve cells (1)

[Kris Knight]

 From a trusted colleague of mine...



> Subject: flu vaccine and nerve cells (1)

>

> Here is a timely review of a recently published article about a  

> connection between Guillain-Barre syndrome, and the HA  

> (hemagglutinin) protein from not only the 1976 swine flu vaccine,  

> but also seasonal vaccines from more recent years.  Apparently, the  

> HA influenza A protein produces antibodies that mimic other  

> molecular antibodies against gangliosides, an important type of  

> molecule very common in the outer membrane of nerve cells.

>

> One has to wonder if this is related to the alarming increase of  

> neurological conditions in humans.

>

> And there is more to this story, which I will post in another email.

>

> Char

> _____________

>

> http://www.newfluwiki2.com/diary/3126/swine-flu-1976-old-vaccines-new-lessons

>

> Anti-Ganglioside Antibody Induction by Swine (A/NJ/1976/H1N1) and  

> Other Influenza Vaccines: Insights into Vaccine-Associated Guillain- 

> Barré Syndrome

>

> The US army suffered heavy losses during the 1918 pandemic - more  

> men died from flu than from fighting in the War.  When an outbreak  

> of swine flu occurred in 1976 among army recruits in a camp, the US  

> government ordered millions of doses of vaccines against the virus,  

> in anticipation of another pandemic.  But a pandemic didn't happen.   

> Instead, the massive vaccination campaign resulted in reports of  

> Guillaine-Barre syndrome (or GBS) in some vaccine recipients,  

> eventually causing the vaccination program to be halted.

>

> Now, decades later, researchers sought to discover how this  

> happened. A paper published last year (link) gives some unexpected  

> and disturbing findings.

>

> SusanC :: Swine Flu 1976 - Old Vaccines, New Lessons?

> First of all, Guillain-Barre Syndrome is an autoimmune neurological  

> disease characterized by acute onset of muscle weakness or  

> paralysis.  Patients develop antibodies to certain molecules (anti- 

> ganglioside antibodies) present in large quantities in nerve tissue,  

> possibly as a result of molecular mimicry ie structural similarity  

> between bacterial or viral antigens and the body's own molecules.

> From the paper:

>

>

> "Patients with GBS develop anti-ganglioside antibodies that are  

> implicated in the pathogenesis of this disease. Antibodies to a  

> number of different complex gangliosides, includingGM1,GD1a, GD3,  

> GT1a, and GQ1b, can be detected in patients with GBS [10].  

> Infections with several agents are associated with the development  

> of GBS [11], and Campylobacter jejuni, a common cause of bacterial  

> gastroenteritis, is one of the most frequently identified bacterial  

> pathogens associated with the development of GBS [12]. It has been  

> deduced that the development of GBS after C. jejuni infection is the  

> result of molecular mimicry between the bacterial surface  

> lipooligosaccharide (LOS) expressing ganglioside-like epitopes and  

> relevant targets in peripheral nerves [10, 13]."

> Because influenza vaccines are grown in eggs, and because eggs are  

> frequently contaminated with C. jejunei, the researchers set out to  

> discover whether vaccine contamination with C. jejunei might be the  

> cause of the GBS outbreak.

>

> Here's what they did.  They obtained unopened vaccine samples from  

> 11 vaccine lots containing the 1976 swine flu vaccine, from 3  

> different manufacturers.  First they tested these vaccines for  

> haemagglutinin inhibition (HAI) test, to prove they still had HA  

> activity.  Then they vaccinated mice with these vaccines, and tested  

> their serum samples for antibodies to C. jejunei.  They did NOT find  

> any such antibodies.  They also ran some PCR tests, again for C.  

> jejunei, with the vaccine samples, and didn't find anything either.

>

> So, that showed them, the 1976 vaccines were NOT contaminated with  

> C. jejunei.  So far so good.

>

> But what they did find was rather disconcerting.  They found that  

> even though no c. jejunei was present, the mice all developed anti- 

> ganglioside antibodies, just like patients with GBS would.

>

> [see chart at above fluwiki link]

>

> After that, they tested 2 other influenza vaccines, from 1991-92,  

> and 2004-05 flu seasons.  They found similar results - the mice did  

> NOT develop antibodies against C. jejunei (showing there was no  

> contamination) but they DID develop anti-ganglioside antibodies.

>

> To verify this in a different way, they did 2 other tests:

>

> 1) they tested the vaccine samples with anti-ganglioside anti-sera,  

> which would be expected to react to the presence of gangliosides or  

> molecules that mimic gangliosides.  They found that the same vaccine  

> lots reacted with these anti-sera, showing that the vaccines indeed  

> contain some substance that acts as ganglioside mimics.

>

> 2) They tested commercial anti-ganglioside anti-sera, for HAI  

> activity, and again found positive results, showing that there may  

> indeed be some mimicry between ganglioside and HA molecults.

>

> So, up to this point, they have shown that there was something in  

> those vaccines that had ganglioside mimic characteristics, and that  

> this may well be the HA molecule itself (since commercial anti- 

> ganglioside anti-sera had HAI activity!).  So far, the tested  

> vaccines were all subunit vaccines.

>

> Finally, they tested a recombinant HA vaccine, ie a vaccine that  

> ONLY contains the HA molecule and nothing else.  This time they used  

> the H5 vaccines, generated against the 1997 Hong Kong virus, and  

> another against the Vietnam 2004 virus.  Again they got the same  

> results - the HA vaccine triggered the development of anti- 

> ganglioside antibodies in vaccinated mice.

>

> [see charts at above fluwiki link]

>

> So what does this study tell us?  If the results are correct (it's  

> always prudent to get other researchers to repeat the study), it  

> shows that the HA in influenza virus itself, irrespective of which  

> virus, has some properties that can trigger anti-ganglioside  

> antibodies.  Although the mice did not develop symptoms of GBS (we  

> know that mice are not good models for human disease) this is a  

> worrying finding.  Current vaccination strategies, whatever vaccine  

> you use, focus on the HA as the main target antigen.  The aim is to  

> produce robust immune response against the HA.  If, as this study  

> suggests, HA is in fact a molecular mimic for molecules in our  

> nervous system, it may not be a smart idea to stimulate strong  

> antibody reactions against HA, after all!

>

> In the words of one FDA scientist, there may be a good reason why we  

> are not MEANT to have strong immune responses against influenza  

> viruses!  Of course, this is speculative at this point, but it  

> really is something worth watching IMO.

>

> _______________________

>

> what the authors said

>

> "Previous studies of the immunogenicity of influenza vaccines in  

> animals or humans have not examined anti-ganglioside antibody  

> responses and should be considered in future vaccine studies."


Kris Knight of WellAware Life Enhancement Center
Phone:  1-608-ALL-LIFE
welaware at merr.com






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